Various hypotheses have been put forward to explain the frequent finding of exocrine insufficiency in diabetes: first, dysregulation of exocrine secretion due to diabetic neuropathy and atrophy of exocrine tissue due to lack of local trophic insulin effects or related to local or general vascular damage; second, simultaneous exocrine and endocrine dysfunction as a net result of a common underlying process affecting the whole pancreas, and autoimmune-mediated inflammation.
Parhatsathid Napatalung from Thailand writes, “The pancreas is harmed if the body is metabolically acid as it tries to maintain bicarbonates. Without sufficient bicarbonates, the pancreas is slowly destroyed, insulin becomes a problem and hence diabetes becomes an issue. Without sufficient bicarbonate buffer, the effect of disease is far reaching as the body becomes acid.”
“Monitoring of blood-sugar levels, insulin production, acid-base balance, and pancreatic bicarbonate and enzyme production before and after test exposures to potentially allergic substances reveals that the pancreas is the first organ to develop inhibited function from varied stresses, writes Dr. William Philpott and Dr. Dwight K. Kalita in their book Brain Allergies.
When one of many possible biological stresses weigh down on the pancreas it will, as any other organ will, begin to function improperly. When this happens the first thing we will see is a reduction in pancreatic bicarbonate production. Once there is an inhibition of pancreatic function and pancreatic bicarbonate flow there naturally follows a chain reaction of inflammatory reactions throughout the body. Under such conditions infections and fungi are known to proliferate.
Decreasing bicarbonate flow would boomerang hardest right back on the pancreas, which itself needs proper alkaline conditions to provide the full amount of bicarbonate necessary for the body.
What oncologists now have to concede is a direct relationship between what is causing diabetes and what is causing cancer; with the diabetic condition itself contributing in a strong way to cancer.