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Selenium

Lipid Replacement Therapy (LRT) can restore and help maintain mitochondrial membrane function by replacing damaged mitochondrial membranes so the perfect form of selenium would have selenium bonded to a lipid. This form was developed by a surgeon in New York who used to inject it to treat cancer.


Lipid Replacement Therapy, the use of functional oral supplements containing cell membrane phospholipids and antioxidants, has been used to replace damaged, usually oxidized, membrane glycerophospholipids in mitochondria that accumulate during aging and in various clinical conditions in order to restore cellular function. We should always remember Dr. Johanna Budwig’s work and diet that provides the best lipids for mitochondrial recovery. 


When selenium is bounded to a lipid, then we do double therapy with one substance, mineral plus Lipid Replacement Therapy (LRT), which is very helpful when our mitochondria are already damaged. When selenium is bound to a lipid the toxicity becomes less than one-thousandth of that of the elements in the forms normally available. So imagine how we can increase the dosage for great but safe effect. 


Selenium improves mitochondrial function even in the absence of oxidative stress. Selenium has beneficial effects of endogenous antioxidant activity via GPx, restoration of mitochondrial function and stimulation of biogenesis, and may also reduce oxidative stress driven inflammation. (See lesson on selenium in part two or Conquering Cancer.)


The most common reasons for a loss of ATP/ADP power include the cell membrane losing its ability to store electrons and/or a depletion of the number of functioning mitochondria. These conditions can be brought about by:


1. Consuming trans or “plastic” fats, which destroy the cell membrane

2. Hypothyroidism, which reduces the number of mitochondria in cells

3. Heavy metals such as lead, mercury, and cadmium

4. Dental infections from decay in teeth, root canals, and in jaw bones

5. Toxins